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<article article-type="research-article" dtd-version="1.3" xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xml:lang="ru"><front><journal-meta><journal-id journal-id-type="publisher-id">medalphabet</journal-id><journal-title-group><journal-title xml:lang="ru">Медицинский алфавит</journal-title><trans-title-group xml:lang="en"><trans-title>Medical alphabet</trans-title></trans-title-group></journal-title-group><issn pub-type="ppub">2078-5631</issn><issn pub-type="epub">2949-2807</issn><publisher><publisher-name>ООО «Альфмед»</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="doi">10.33667/2078-5631-2022-17-15-18</article-id><article-id custom-type="elpub" pub-id-type="custom">medalphabet-2745</article-id><article-categories><subj-group subj-group-type="heading"><subject>Research Article</subject></subj-group><subj-group subj-group-type="section-heading" xml:lang="ru"><subject>Статьи</subject></subj-group></article-categories><title-group><article-title>Варианты течения хронической сердечной недостаточности у больных с сердечно-сосудистой патологией после перенесенной коронавирусной инфекции</article-title><trans-title-group xml:lang="en"><trans-title>Variants of course of chronic heart failure in patients with cardiovascular pathology after coronavirus infection</trans-title></trans-title-group></title-group><contrib-group><contrib contrib-type="author" corresp="yes"><contrib-id contrib-id-type="orcid">https://orcid.org/0000-0003-0250-0052</contrib-id><name-alternatives><name name-style="eastern" xml:lang="ru"><surname>Панина</surname><given-names>Ю. Н.</given-names></name><name name-style="western" xml:lang="en"><surname>Panina</surname><given-names>Yu. N.</given-names></name></name-alternatives><bio xml:lang="ru"><p>Юлия Николаевна Панина, к. м. н., доцент</p><p>кафедра внутренних болезней</p><p>Орел</p></bio><bio xml:lang="en"><p>Iuliia N. Panina, PhD Med, associate professor</p><p>Dept of Internal Diseases</p><p>Orel</p></bio><email xlink:type="simple">doc.panina@yandex.ru</email><xref ref-type="aff" rid="aff-1"/></contrib><contrib contrib-type="author" corresp="yes"><contrib-id contrib-id-type="orcid">https://orcid.org/0000-0001-5406-7065</contrib-id><name-alternatives><name name-style="eastern" xml:lang="ru"><surname>Вишневский</surname><given-names>В. И.</given-names></name><name name-style="western" xml:lang="en"><surname>Vishnevskii</surname><given-names>V. I.</given-names></name></name-alternatives><bio xml:lang="ru"><p>Валерий Иванович Вишневский, д. м. н., проф., зав. кафедрой</p><p>кафедра внутренних болезней</p><p>Орел</p></bio><bio xml:lang="en"><p>Valerii I. Vishnevskii, professor, holder of advanced doctorate in medical sciences, head of Dept</p><p>Dept of Internal Diseases</p><p>Orel</p></bio><xref ref-type="aff" rid="aff-1"/></contrib></contrib-group><aff-alternatives id="aff-1"><aff xml:lang="ru"><institution>Медицинский институт ФГБОУ ВО «Орловский государственный университет имени И. С. Тургенева»</institution><country>Россия</country></aff><aff xml:lang="en"><institution>Orel State University n. a. I. S. Turgenev</institution><country>Russian Federation</country></aff></aff-alternatives><pub-date pub-type="collection"><year>2022</year></pub-date><pub-date pub-type="epub"><day>07</day><month>09</month><year>2022</year></pub-date><volume>0</volume><issue>17</issue><issue-title>Кардиология. Неотложная медицина (2)</issue-title><fpage>15</fpage><lpage>18</lpage><permissions><copyright-statement>Copyright &amp;#x00A9; Панина Ю.Н., Вишневский В.И., 2022</copyright-statement><copyright-year>2022</copyright-year><copyright-holder xml:lang="ru">Панина Ю.Н., Вишневский В.И.</copyright-holder><copyright-holder xml:lang="en">Panina Y.N., Vishnevskii V.I.</copyright-holder><license xml:lang="ru" license-type="creative-commons-attribution" xlink:href="https://creativecommons.org/licenses/by/4.0/" xlink:type="simple"><license-p>Данная работа распространяется под лицензией Creative Commons Attribution 4.0.</license-p></license><license xml:lang="en" license-type="creative-commons-attribution" xlink:href="https://creativecommons.org/licenses/by/4.0/" xlink:type="simple"><license-p>This work is licensed under a Creative Commons Attribution 4.0 License.</license-p></license></permissions><self-uri xlink:href="https://www.med-alphabet.com/jour/article/view/2745">https://www.med-alphabet.com/jour/article/view/2745</self-uri><abstract><p>   В статье проанализированы значимые для клиницистов исследования, направленные на понимание этиопатогенетических механизмов, которые способствуют развитию ишемической, неишемической и вызванной вирусами или бактериями сердечной недостаточности. Проведен систематический поиск литературы за 2019–2021 годы в PubMed, EuropePMC, SCOPUS, Кокрановской центральной базе данных и medRxiv, использованы поисковые запросы «сердечная недостаточность» и COVID-19, затем мы сконцентрировались на публикациях, которые содержат данные о вариантах течения хронической сердечной недостаточности у больных с сердечно-сосудистой патологией после перенесенной коронавирусной инфекции и провели их анализ с последующим теоретическим моделированием. Распространенность хронической сердечной недостаточности среди населения, восприимчивого к COVID-19, значительна, равно как и распространенность предрасполагающих состояний, которые подвергают инфицированных пациентов риску развития сердечной недостаточности во время болезни и в отдаленном периоде после выздоровления. По этой причине распространение глубоких знаний о клинических последствиях и прогностическом влиянии COVID-19 на эту уязвимую категорию является приоритетом.</p></abstract><trans-abstract xml:lang="en"><p>   This article analyzes studies that are significant for clinicians aimed at understanding the etiopathogenetic mechanisms that contribute to the development of ischemic, non-ischemic and viral/bacterial-induced heart failure. A systematic search of literature for 2019–2021 was conducted in PubMed, EuropePMC, SCOPUS, the Cochrane Central Database and medRxiv using the search queries ‘Heart Failure’ and ‘COVID-19’; then we focused on publications that contain data on variants of the course of chronic heart failure in patients with cardiovascular pathology after a coronavirus infection and analyzed them with subsequent theoretical modeling. The prevalence of chronic heart failure among the population susceptible to COVID-19 is significant, as is the prevalence of predisposing conditions that expose infected patients to the risk of developing heart failure during illness and in the long-term period after recovery. For this reason, the dissemination of in-depth knowledge about the clinical consequences and prognostic impact of COVID-19 on this vulnerable category is a priority.</p></trans-abstract><kwd-group xml:lang="ru"><kwd>хроническая сердечная недостаточность</kwd><kwd>COVID-19</kwd><kwd>ремоделирование сердца</kwd><kwd>биомаркеры</kwd><kwd>фиброз миокарда</kwd><kwd>отсроченные послеинфекционные осложнения</kwd></kwd-group><kwd-group xml:lang="en"><kwd>chronic heart failure</kwd><kwd>COVID-19</kwd><kwd>cardiac remodeling</kwd><kwd>biomarkers</kwd><kwd>myocardial fibrosis</kwd><kwd>delayed postinfectious complications</kwd></kwd-group></article-meta></front><back><ref-list><title>References</title><ref id="cit1"><label>1</label><citation-alternatives><mixed-citation xml:lang="ru">Alon D. et al. Predictors and outcomes of infection-related hospital admissions of heart failure patients. PloS one 2013; 8: e72476.</mixed-citation><mixed-citation xml:lang="en">Alon D. et al. 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